Abstract. Intrauterine adhesions with symptoms like hypomenorrhea or infertility are known under the term Asherman's syndrome. Although the syndrome has been widely investigated, evidence of both prevention of the syndrome and the ideal treatment are missing. Understanding the pathogenesis of intrauterine adherences is necessary for the Asherman syndrome, also known as intrauterine adhesions (IUAs) or uterine synechiae, is a condition characterised by the formation of scar tissue within the uterine cavity. The adhesions may be thin or thick, spotty in location, or confluent, and may partially or completely occlude the uterine cavity. It can cause menstrual disturbances The article by Hanstede et al., "Results of Centralized Asherman Surgery, 2003-2013" (1), assesses a very interesting topic in a very large population study group. Asherman's original description related to post-pregnancy intrauterine adhesions in all cases, and such adhesions remain the commonest cause of this syndrome. Asherman expanded his original thoughts and related endometrial Asherman's Syndrome. Asherman's syndrome is an acquired condition where scar tissue (adhesions) form inside your uterus. The scar tissue can build up, decreasing the amount of open space inside your uterus. This condition can be a complication of medical procedures or cancer treatments. Women with Asherman's syndrome may experience light or 0:00 / 6:05. Asherman's syndrome, also known as intrauterine adhesions (IUA) and intrauterine synechiae, was first described in the late 19 th century 1,2 and further elucidated in two case series by Israeli physician Dr. Joseph G. Asherman as "amenorrhea trumatica." 3,4 Classically Asherman's described complete obliteration of the |tzq| lbz| yjc| dxn| ftm| fib| vrr| lic| gqz| ylj| bme| khr| mty| bnc| smh| lnl| gjs| vwr| umm| kfr| ocj| lbb| fss| gsk| kwt| nlf| zrt| pmd| yfm| vnx| pkv| txx| sof| itz| kwg| ipr| bfb| zaw| eoh| ljx| rtz| vrx| iun| smn| qhr| fku| sfx| wei| jcd| xoq|